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The underlying explanation remains unclear
The activation of AMPK helps prevent the occurrence of ferroptosis, while its inhibition increases the risk of ferroptosis
This contraindication stems from observations in rodent studies where semaglutide and other GLP-1 receptor agonists caused dose-dependent and treatment-duration-dependent thyroid C-cell tumors
Continued research and clinical trials are essential to fully understand and utilize BPC 157s therapeutic potential in this area
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