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GLP-1 plays a key role in maintaining glucose homeostasis, as it markedly increases glucose-stimulated insulin secretion (GSIS) (77) and attenuates hepatic glucose production, independent of its effect on pancreatic islets (78, 79)

In research models, the without-DAC form (Mod GRF 1-29) is described as producing short GH peaks within 1530 minutes, mimicking the endogenous secretion rhythm

Therefore, we hypothesized that a 1-wk treatment dose of IGF-1 LR3 into FGR fetal sheep produced using a model of severe placental insufficiency that replicates features of human placental insufficiency and FGR (6, 7, 27) would improve growth without further lowering insulin concentrations or GSIS