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These effects typically resolve within hours and decrease with continued use as skin adapts
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A Multi-Pathway Model We can think of this as a convergence of several mechanisms: L-cell activation from viral or microbial sensing Vagal signaling altered by cytokine release Gut-brain axis dysregulation, mirroring GLP-1 agonist effects Delayed motility from enteric nervous system inflammation Together, these pathways could produce a temporary GLP-1-like state during and shortly after the viral illness
In addition to appetite suppression, semaglutide GLP-1 slows gastric emptying, meaning that food remains in the stomach longer, enhancing the feeling of satiety after meals
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