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Timing requirements for insulin/IGF-1 signaling in C

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Figure 1 2.4.1 Association between microplastics and CNS metabolic disorders Microplastics contribute to neuronal dysfunction by interfering with metabolic pathways,for example, microplastic exposure may lead to the development of insulin resistance and metabolic syndrome (26)

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Across preclinical studies, Epitalon has shown: Telomerase activation and telomere extension: Via TRAP assays and fibroblast lifespan extension beyond the Hayflick limit Epigenetic remodeling: Reversal of chromatin aging, histone modulation, and gene promoter binding Redox homeostasis: Increased antioxidant enzymes (SOD, NQO1, catalase) and reduced ROS, lipid peroxidation, and DNA damage Circadian restoration: Upregulation of AANAT/pCREB, normalized melatonin and cortisol rhythms, and clock gene modulation in humans Cancer risk reduction: Reduced tumor multiplicity and delayed onset in HER-2/neu and colon cancer models