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Vitamin B-6-induced neuropathy: exploring the mechanisms of pyridoxine toxicity

Inactivation of the Cardiomyocyte Glucagon-Like Peptide-1 Receptor (GLP-1R) Unmasks Cardiomyocyte-Independent GLP-1R-Mediated Cardioprotection

The pathological increase in NADPH oxidase-mediated superoxide (O) and peroxide production from inflammatory phagocytic cells in the course of this inflammatory reaction acts as source of oxidative stress

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