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Glucagon-Like Peptide-1 Receptor Agonists and Age-Related Macular Degeneration
In the present review, we described the key steps in hepatic fat metabolism, from synthesis to catabolism by lipolysis and lipophagy and discussed potential steps where alterations in the normal pathways might contribute to the development and progression of NAFLD, with a special focus on recent reports describing that an acquired impairment in LAL activity may be implicated in NAFLD pathogenesis
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