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This was also detected by GO term enrichment of response to oxidative stress and glutathione metabolic process (Supplementary Fig

Mostly importantly, our findings should motivate longitudinal studies using 7T MRS to follow neurochemical changes in individual patents across the phases of their illness

For in-depth reviews focused on tumor progression and its impact on the BBB, please see Dubois et al

PMID: 41203627 Acta Pharmacol Sin Targeting PPAR activation sensitizes glioblastoma cells to temozolomide and reverses acquired resistance by inhibiting H3K18 lactylation

overexpressed miR-340-5p in the myocardium by direct myocardial injection of AVV-9 containing miR-340-5p precursor, which targeted inhibition of MyD88 and thus restored declining GSH levels, alleviated insult of cardiac function and oxidative injury induced by sepsis in the hearts of mice [253]