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Context Diabetes mellitus (DM) is a rapidly increasing chronic metabolic disorder that poses a significant threat to global health

Interventions that decreased or increased PPAR- activity (including exposure to a thiazolidinedione) resulted in corresponding changes in GIP receptor expression in in vivo rat models

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This timeframe allows the medication's effects on gastric motility to diminish while minimizing any rebound side effects or loss of metabolic benefit