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Campone, M., et al
It is thus tempting to suggest that in addition to modulating GLP-1R desensitization and thus the GLP-1R/Gs/cAMP axis, GRK2 may regulate GLP-1R-dependent insulin release by sequestering -arrestin1 away from EPAC2 (see scheme in Fig
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Importantly, this effect often attenuates over time with long-acting agents (tachyphylaxis), though the weight loss and glycemic benefits typically persist
From screening to treatment: the new landscape of diabetic kidney disease
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