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Aging reduces NAD+ synthesis pathways while increasing consumption by enzymes like CD38 and PARPs, activated by inflammation and damage.[13][15] CD38, an immune-related enzyme, ramps up with age, breaking down NAD+ precursors

This dual action makes it valuable for studying tissue engineering applications and investigating cartilage repair mechanisms in degenerative joint conditions (4)

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Though generally safe, following the recommended dosage guidelines is crucial for avoiding potential negative side consequences