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Bioinformatics and in silico genomic analyses have identified a catalog of small molecules as potential regulators of SVZ microdomain-specific lineages (Azim et al., Alternative Roles of SVZ-Derived Progenitors in Myelin Repair Martino's laboratory investigated the role of SVZ-derived progenitors in myelin repair, using nestin-thymidine kinase-transgenic mice to kill neural progenitors in a specific manner in cuprizone-fed mice (Butti et al., Thus, endogenous SVZ progenitors may promote myelin repair via two different mechanisms: OLG replacement and immunomodulation/neuroprotection

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