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Most of the mitochondrial oxygen is utilized for ATP production by the mitochondrial electron transport chain (ETC) through oxidative phosphorylation (OXPHOS) [18]

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Importantly, this relationship is relevant to humans as individuals with exceptional longevity are enriched with functional IGF-1R mutations 13 , while low IGF-1 levels predict better survival in female nonagenarians 16

This process comprises encapsulating debris in double-membrane autophagosomes, followed by fusion of autophagosomes with lysosomes [248]