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While the initial, weakly-desensitized AMPAR-like state for wild-type KAR is currently not available, it is most likely similar to the state represented by the recent mutant GluK2 structure (K676C and N802C) in the agonist-bound non-conducting state trapped by disulfide crosslinking across the LBD dimer interfaces (Zhou et al., 2025) (Figure 8A)

A permissive apoptotic environment: function of a decrease in intracellular superoxide anion and cytosolic acidification

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