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doi:10.1007/s00726-009-0269-0 [Pubmed] [Springer] T J de Koning
BPC-157 is believed to interact with the nitric oxide (NO) system, angiogenic growth factors, and cellular signaling pathways like FAK-paxillin and VEGF, which are involved in cell migration, cytoskeletal reorganization, and vascular modulation
GHRP-6 also raises cortisol and prolactin levels significantly
The proposed mechanism involves PCs activating peroxisome proliferator-activated receptor (PPAR) in macrophages, upregulating PPAR target genes (CD36 and ABCG1) and driving the transition from M1 to M2 phenotypes
doi: 10.1186/1475-2891-13-11 268
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