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Dysbiosis of the microbiome forms a new internal environment, under which it may affect the development of GERD from the perspectives of molecular mechanisms: microbial activation of Toll-like receptors, microbial stimulation of cyclooxygenase-2 expression, microbial stimulation of inducible nitrous oxide synthase, and activation of the NLRP3 inflammatory vesicle

The large-scale manufacturing of medicines through compounding means that these products are being provided in large quantities, without the regulation and evaluation that such volumes should undergo

Source: Traverso, N., Ricciarelli, R., Nitti, M., et al

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