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These results highlight hepsin's crucial role in controlling APAP-induced liver toxicity by regulating the cellular abundance of GJ proteins, which can affect the transmission of toxic metabolites or cell death signals and potentially increase oxidative stress
Figure 2 The viability results of frozen-thawed spermatozoa, shown in Figure 2B, were partially consistent with the motility results of frozen-thawed spermatozoa as viability in the GSH + SeNPs group (69.44 3.24%) was significantly higher than that in the NC (56.33 7.43%), GSH (61.89 4.35%) and SeNPs (63.49 3.23%) groups ( P 0.05)
Moreover, CRC cells exhibit marked metabolic plasticity, in which FAO serves as a key mitochondrial mechanism for energy compensation [13, 14]
Beyond 12 weeks, cumulative compounding occurs
Labunskyy, V
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