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Although the impact of GLP-1RAs on pancreatic cancer risk remains controversial, emerging evidence suggests that chronic low-grade inflammation and proliferative changes may constitute key pathophysiological mechanisms underlying pancreatic cancer development in patients receiving GLP-1RA therapy [8]
MSphere 5, e00183e00120
Its role lies in genomic maintenance and early-stage risk reduction, not tumor eradication
When poorly matched observational data were corrected for these variables, the apparent association disappeared
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