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One mouse was found dead in vehicle group

GSH-deficient FoB upregulate glycolysis but accumulate defective mitochondria Based on our scRNA analyses and the effect of Gclc deficiency on glucose uptake (Fig

Almost simultaneously, these cells showed a significantly increased cellular efficiency to produce ROS after stimulation, as determined by an SI calculated from the unstimulated and stimulated fluorescence intensity of ROS-producing PMN

GPX4 is not essential for T cell development, but plays a crucial role in maintaining T cell homeostasis and supporting T cell-dependent immune responses during acute viral infections [59]