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CzEcp2, CzNIS1a and CzNIS1b were subsequently transiently expressed in model plant systems and shown to trigger non-host resistance, suggesting they target conserved structures or processes to facilitate pathogen colonization and growth during the switch to necrotrophy
A.SainsburyA.BoeyD.HerzogH.et al (2005)
Finally, elevated cAMP concentrations, in combination with Ex-4, activate the p38 signaling pathway in breast cancer cells, resulting in decreased cell growth and increased apoptosis [58]
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