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J Clin Med 2024;13:6
is supported by the Leicester NIHR Leicester Biomedical Research Centre and the University of Leicester
If NAD+ is depleted, which caloric restriction can cause, the efficiency of these metabolic transitions suffers
Despite localization of mGIPR-Ab/P3 predominantly in CVOs, and not in deeper parenchyma of the brain, c-Fos activation was observed in multiple neural substrates involved in food intake control 21,22,23,24,25,26,27,28,29,30 (Fig
Combining these effects with severe caloric restriction could exacerbate gastrointestinal distress and increase the risk of dehydration
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