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Writing and rewriting the epigenetic code of cancer cells: from engineered proteins to small molecules

An update on chronic complications of diabetes mellitus: from molecular mechanisms to therapeutic strategies with a focus on metabolic memory

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10.3390/nu16162753 30 FerreiraB.MendesF.OsrioN.CaseiroA.GabrielA.ValadoA

Such stress-associated -cell states are known to display impaired incretin responsiveness due to reduced GLP1R signaling and diminished cAMP pathway activation (Luo et al., 2013)[Gao], suggesting that inflammatory axes linked to dysbiosis may differentially affect vulnerable -cell subpopulations