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An increase in the levels of both thiols following GSH treatment promoted both infection and extension of defense responses in sym33-3 nodules, whereas a similar increase in sym40-1 nodules led to the formation of infected cells resembling wild-type nitrogen-fixing cells and the disappearance of an early senescence zone in the base of the nodule
Neutropenia raises the risk of life-threatening infection and can interrupt treatment schedules (Crawford 2004
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In summary, enhancing mitochondrial biogenesis through gene therapy can be achieved either by delivering genes like PGC-1 and TFAM via AAV, or by regulating the expression of relevant factors in monocytes through preoperative ex vivo lentiviral transfectionboth contributing to preconditioning for elective surgeries
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