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Other intermembrane proteins released into the cytosol such as cytochrome c and Smac/DIABLO, do not promote apoptosis because APAP-induced mitochondrial dysfunction and oxidant stress prevents any apoptotic signaling in hepatocytes [54, 55]
These properties underpin MT3s critical role in neuronal copper buffering and oxidative defense (150, 163)
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For groups iii and iv, the dosages of AuNPs and Phe-BF 3 were 5 mg kg 1 and 30 mg kg 1 , respectively
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