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This upregulation enhances cellular responsiveness to growth hormone, potentially amplifying endogenous growth factor signaling critical for tissue regeneration
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Researchers noted this reduction exceeded what weight loss alone would predict, suggesting direct hepatic effects of glucagon receptor agonism including enhanced fatty acid oxidation
Crucially, growing preclinical data emphasize that radiation response is histology-dependent and that radiosensitization strategies may therefore yield differential benefit across tumor subtypes [171, 183]
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