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SOD, GSH and catalase are dominating intracellular antioxidants present or released into the circulation following disease related erythrocyte catastrophe21, their plasma/serum activities are changed by several orders of magnitude than corresponding erythrocyte levels22

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In addition, Caveolin-1 (Cav-1), a 21-kDa integral plasma membrane protein is a well-known negative regulator of eNOS activity in vascular endothelial cells 17,18,19,20,21,22