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Targeting early degenerative changes, such as synaptic loss, axonal transport disruption, mitochondrial compromise, and glial dysfunction, may prove more effective than attempting to reverse advanced neuronal death [27, 28]

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BAPTA-AM is activated by intracellular esterases to form BAPTA, which specifically chelates intracellular free Ca, promoting the restoration of calcium homeostasis and inhibiting endoplasmic reticulum stress and calcium-dependent apoptosis pathways