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In models of heart failure (HF), GLP-1 RAs have been observed to alter energy substrate utilization, alleviate myocardial and endothelial inflammation, mitigate oxidative stress, and antagonize myocardial remodeling [14]
"Kinetics, pharmacokinetics, and regulation of L-carnitine and acetyl-L-carnitine metabolism." Ann NY Acad Sci 2004;1033:30-41
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Early findings are encouraging for instance, selectively inhibiting the NLRP3 inflammasome or scavenging circulating mtDNA in animal models markedly attenuates IR-induced organ damage (27)
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