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Anyone describing a precise day-by-day injectable GHK-Cu onset with confidence is extrapolating beyond available data

Nerve conduction studies or electromyography are generally reserved for atypical presentations (such as marked asymmetry, predominant motor involvement, or rapid progression), diagnostic uncertainty, or when other causes are suspected

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The reasoning is that oral bioavailability is ~4050%, and animal study HED conversions suggest higher doses may be needed for the full effect

GHRP-2, being a synthetic ghrelin agonist, effectively fools the body into thinking theres more ghrelin present