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Indian Journal of Dermatology, Venereology and Leprology.

Picture rheumatoid arthritis as a fortress besieged by its own guards: a T-cell mediated autoimmune disease where the immune system, mistaking joint linings for foreign foes, launches unprovoked assaults [4]

Nonetheless, other genes recently discovered are being studied to determine the contribution to the disease onset and development (Shen et al

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However, p53 mutations/deletions in CRC cells enhance glycolytic dependency, driving cell cycle progression (G2/M G1 transition) and energy production predominantly through glycolysis [66]