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The cascade of events triggered by glucose excretion seems very complex, interlacing with inflammation, lipolysis, and deeper intracellular changes, so it might take a long time to solve it completely

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More specifically, the ARC nucleus of the hypothalamus has been highlighted as one of the main sites mediating this action as chemical lesion of the ARC in rats ablates the anorectic action of GLP-1 (Tang-Christensen et al., 1998)

Given that the main pathophysiological features of GIH include central and peripheral insulin resistance, as well as -cell dysfunction, it is reasonable to infer that insulin sensitizers and incretin-based agents could complement the use of insulin or secretagogues

Store at 28C, protect from light, do not freeze