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Metabolic analysis revealed that the source of the anti-ferroptosis effect of GCH1-overexpressing cells was BH4/BH2, supplementation with which rescued cell viability
Molybdenum-bridged endo-exogenous antioxidant synergy reverses acute kidney injury via mitochondrial homeostasis reconstruction
Montero-Melendez, K
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Since chemokine receptors are key downstream effectors of angiogenesis induction, we evaluated the regulation of CXCR4, CXCR1, CXCR2, CCR2 expression on endothelial cells, incubated with conditioned medium from ALCAR treated PCa and BPH cell lines
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