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demonstrated in 2001 that AOD-9604 stimulates lipolysis in adipose tissue without activating the growth hormone receptor, a finding that separated its fat-metabolizing action from GH-related proliferative risks [1]
Despite this potential, given the known long-term benefit of lowering hemoglobin A1c in preventing DR progression, there does not appear to be any contraindication to initiating GLP-1/GIP agonist therapy with appropriate ocular monitoring
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