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A 2017 study was the first to demonstrate activation of ferroptotic pathways following ICH (183), and subsequent studies have confirmed that ferroptosis contributes to neuronal damage after hemorrhage via mechanisms involving neurotoxin release and oxidative stress (184, 185)
Smooth muscle cells and the formation, degeneration, and rupture of saccular intracranial aneurysm wall--a review of current pathophysiological knowledge
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Additional information on materials and methods can be found in the Supplementary Materials and Methods
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