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Over 100 preclinical studies document tissue repair, gastrointestinal protection, wound healing, and neuroprotective effects

Yes, if the E/M service is significant and separately identifiable from the injection

Research from a 2023 article found significant mitochondrial structural and functional abnormalities in the muscle cells of long COVID patients, including: Disrupted cristae (mitochondrial) architecture Impaired electron transport chain function Reduced ATP production capacity These findings mirror what we see in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), a condition that shares significant overlap with long COVID and is now thought to involve similar mitochondrial pathology

This leaves you open to oxidative damage caused by reactive oxygen species (aka free radicals)

246 For example, SMAD2 ubiquitination by Itch/AIP4 promotes its phosphorylation by TRI, facilitating signal transduction