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Following DNA damage, the cell cycle can be transiently arrested to allow for DNA repair or activation of pathways leading to programmed cell death (apoptosis) when the damage cannot be repaired (29)

PubMed PMID: 9403790

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This is consistent with our earlier reports where the mitochondrial damage due to GNE mutation could not be reverted by sialic acid supplementation alone 16

Several studies have shown that IGF-2 in the tumor microenvironment is derived mainly from CAFs and that high levels of IGF-2 inhibit the infiltration and cytotoxicity of CD8+ T cells, exacerbating the immunosuppressive effect of tumors (78,141)