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These findings suggest that repetitive element methylation may be less sensitive or not consistently altered by NS exposure compared to specific gene methylation 227
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Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired
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