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Ex-4 also promotes brain-derived insulin production and enhances insulin signaling through the Wnt/-catenin/NeuroD1 signaling pathway, which subsequently reduces tau hyperphosphorylation and cognitive dysfunction in diabetic mice [64]
An Israeli nationwide cohort of IBD patients (Epi-IIRN) included 633 patients with concomitant DM who received GLP1RA for at least six months
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