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The fact that GSH depletion is one of the earliest brain abnormalities in Parkinsons has led to several studies assessing the effects of intranasal GSH supplementation in that disease

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Animal models of depression show a decrease in cell proliferation (creation of new brain cells) as well as an increase in cell death (apoptosis).[ref] A relative lack of ATP due to mitochondrial dysfunction could also impact the ability of neurotransmitters to bind to their receptors

10.1186/s13054-019-2548-9 216 ZhangQ.RaoofM.ChenY.SumiY.SursalT.JungerW.et al (2010)