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The underlying mechanism linking hyperhomocysteinemia and dementia is the development of endothelial function and impairment of cerebral blood flow with subsequent oxidative stress-induced neuronal injury (Kovalska et al

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In this paper, we consider as outcomes of interest the [GSH]/[GSSG] ratio and the model outputs for the concentration of NADPH, ROS and G6P at time points for which calibration and validation data measurements are available (PN12, PN13, PN15, PN17, PN28)

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