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Cagrilintide is engineered to have enhanced receptor affinity and a significantly prolonged half-life compared to native amylin, allowing for more sustained biological activity in experimental models
Although the knockout of GPX7 does not lead to the direct death of mice, the production of excess reactive oxygen species leads to incorrect oxidative modification of proteins and irreversible changes in function, resulting in various diseases
The decrease is directly proportional to the GPx activity in the sample
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