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GSH serves as a substrate for glutathione peroxidases and glutathione S-transferases and participates in reversible protein S-glutathionylation (Lu, 2013)
Most studies leverage joint-cavity acidification for targeted delivery of mitochondrial antioxidants, metabolic modulators, or autophagy-promoting molecules to diseased chondrocytes to restore mitochondrial homeostasis
Understanding complementary mechanisms guides effective combination strategies
Science 373 , eabd2893 (2021)
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