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Bray GA, et al
Role of nrf2 in oxidative stress and toxicity
In summer, evening application becomes even more important
We propose that inhibiting glycolysis and the fatty acid synthesis (FAS) pathway in M1 macrophages, or enhancing fatty acid oxidation (FAO) in M2 macrophages, may effectively reduce foam cell formation, mitigate inflammation, and slow the progression of atherosclerosis ( 6 Clinical translation and therapeutic perspectives While numerous metabolic targets (e.g., PKM2, PDK4, IDO, CPT1) and modulators (e.g., DMF, itaconate, rapamycin) show promise in preclinical models, significant barriers remain in translating these findings into clinical therapies
Research shows it can protect esophageal tissue from acid damage and promote healing of esophageal lesions
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