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It can help reduce oxidative damage to the brain and support overall cognitive function

Hence, when TD is limited to the gastrointestinal system, the pathology-relevant perturbation of ACh signaling may rather be expected in the enteric nervous system (ENS)

Improve metabolic health by reducing adiposity, sustaining muscle function, and optimizing glucose regulation [2][5][8]

Improved cell health translates directly to improved sperm concentration and progressive motility results

Preclinical models demonstrate that region-specific elevations of central KYNA highlight a functional trade-off: while KYNA is often considered neuroprotective due to its antagonism of glutamatergic receptors, its broad receptor profile also allows it to inhibit 7nAChRs, which are essential for prefrontal cortical processing and mood regulation