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Since BPC-157 is derived from a gastric protein, it has a natural affinity for the gastrointestinal tract
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13a, Supplementary Table 9), suggesting that the distinct rotamer orientation of H296 ECL2 in Cagri-bound CTR (to accommodate the Glu linker) and/or the observed more open ECL3 conformation induced by the lipidated peptide may facilitate or stabilize an active CTR conformation (Supplementary Fig
The mechanisms overlap completely, and combining them would dramatically increase gastrointestinal side effects without meaningful additional benefit
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