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Thus, we speculate that T3 can increase the production of non-12-OH FXR-antagonistic BAs in human likely attributed to the same mechanisms in mice, thereby promoting GLP-1 and insulin production by suppressing intestinal FXR signaling
Based on route of administration, the parenteral segment is projected to dominate in 2026
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Constantinescu, A., Tritschler, H., and Packer, L
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