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The two pathways converge on somatotroph GH release through independent second-messenger cascades, and the combined-receptor pharmacology in healthy-adult human preparations across the Bowers laboratory work in the 1980s and 1990s reported a larger GH excursion under combined GHRH-analog plus GHRP-class administration than under either compound singly [6]

The first is extracellular matrix biology: fibroblast culture and rat wound-chamber work from the Maquart and Simeon groups reported that GHK-Cu stimulates collagen, glycosaminoglycan and small proteoglycan synthesis while simultaneously upregulating matrix metalloproteinase-2 and its tissue inhibitors, a combined synthesis-and-turnover signature unusual among matrix-active compounds

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While direct, peer-reviewed, head-to-head clinical trials comparing the two forms are still emerging, we can extrapolate a great deal from the known chemical properties

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