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TRIM28-mediated nuclear retention of TRIM37 is essential for PGC development To further investigate the interaction between TRIM37 and TRIM28, we overexpressed TRIM37 in HeLa cells and observed that the majority of TRIM37 localized to the cytoplasm (Fig

In SMMC7721 and QGY7701 HCC cells, QUE promoted the expression of p53, inhibited Bcl-xl, and up-regulated the expression of Caspase-3/9, which enhanced the effect of DOX on promoting apoptosis (54)

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It would also need a plan for monitoring side effects and stopping criteria