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Loss of GSH synthesis resulted in a decreased expression of lipogenic enzymes, including SCD1

Studies have shown that JNK, when combined with Sab, which is a scaffold protein located in the outer membrane of mitochondria, triggers the destruction of mitochondrial electron transport chain and promotes the release of reactive oxygen species (ROS), ultimately resulting in cell death (71, 72)

**Setria also appears to help maintain high NO levels, which may reduce fatigue and speed recovery

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