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By using a reducible disulfide linker, we built in a redox-sensitive release mechanism to facilitate intracellular release of the unmodified NMDA receptor antagonist, enabling the combined cellular actions of GLP-1 receptor agonism and NMDA receptor antagonism

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Pathophysiology of increased nuchal translucency: a review of the literature

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True enzymatic rates were obtained by subtracting the non-enzymatic rates (no enzyme present) from the total rates given by these experiments