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Multiple cellular processes are considered to contribute to this functional decline and these are collectively referred to as the hallmarks of aging and these include processes like genomic instability, telomere attrition, epigenetic alterations, loss of proteostasis, disabled macroautophagy, deregulated nutrient-sensing, mitochondrial dysfunction, cellular senescence, stem cell exhaustion, altered intercellular communication, chronic inflammation, and dysbiosis 1

Proposed actions include modulation of caspase-3 activity and influence over cytokines such as IL-6 and TNF-, which are often associated with inflammatory states

Changing in pH and salt concentration will alter electrostatic interactions between charged amino acids

Over 100 peer-reviewed studies have been published on BPC-157, the large majority in rodent models

Do not allow working stock to age beyond the stability window